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Vitamin E and cancer: Be careful with improvements!

One study shows that the enhancement of vitamin E can increase the risk of cancer in people with specific genes.

Improved advances

Increased nutrition, especially vitamin, is still highly regarded, with almost half of the Canadian eating daily. This fish is very spectacular, because the results of a number of clinical studies are unsure: people who use antioxidant improvements, whether complex, vitamin C or E, or beta -carotene, a & # 39; shows that there is no reduction in cardiovascular or cancer ailments (1).

Not only are those provisions ineffective, but some of the checks have been recorded by a number of negative effects associated with a & # 39; wearing a lot of dolls. Vitamine E vitamin enhancements are particularly difficult, with a marked increase in the risk of lung cancer when combined with beta-carotene (in smoking), a significant increase in the risk of cancer prostain, and a slight increase in improve cancer risk. die too quickly. In a cancer case, high vitamin sites with high antioxidant activity (such as patient E) appear to eliminate a median balance between free radicals created by cells and antioxidant protections natural nature (2). So instead of preventing cancer, large doses of antioxidants can progressively improve the development of this disease by blocking normal work; body, and include the systems that are involved in a & # 39; eliminate your toothpasts.

Different variations

Recent study has shown that the impact of vitamin E on the risk of cancer is highly dependent on the genetic background of the people who are experiencing the risk of cancer. using the developments (3). Using the data obtained during the Women's Genom Health Check (23,294 partners) and the Alpha-Tocopherol Caretene Food Abduction Testing (29,133 participants), researchers investigated most of their clients; Cancer in these groups is based on genetic changes in genealogy; coding-catechol-O-methyltransferase (COMT), an engine that was involved in vitamin E. metabolism. Remember that we have two (alleles) versions of each gene, and these versions can be the same (homozygotes) or different (heterozygotes). For example, in the case of a TSE genealogy that was inspected in this study, there are two main homozygous changes called meeting / meeting and val / val and different heterozagous (val / met).

The results obtained show a significant difference in cancer risk according to the nature of the COMT genealogy generations found in the partners in both inspections. In individuals with the var / homo variables homozygous, the number of vitamin E foods increases the total risk of cancer by 15% compared to placebo, but at risk / manzygotes jobs this risk is reduced by 14%. There is no effect on vitamin E in the heterozygotes val / met.

The blue / performance numbers and val / val homozygotes represent about 25% of the population, and the rest; taking the heterozygous form of the COMT gene. In practice, vitamin E has a beneficial effect of vitamin E in three-quarters of people, with a quarter of which is even It is at higher risk of cancer. The benefits of Vitamin E access only met / met homozygotes, representing about 25% of the population. Since we are not aware of the changes of the COMT generation that our parents leave us, gives a dangerous promise to increase vitamin E!

Non-essential risk

It is easier to find a risk of getting vitamin E with a good diet. Vegetable oils, nuts (and peanuts) and some fruit and vegetables such as avocados, spinach or asparagus are all very good sources of sweetness and support the needs of cancer and enough to prevent cancer.

(1) Bjelakovic G et al. Reduced and diecial preparations. Clinic Curric Clinic Clinic Description Description. 2014; 17: 40-44. (2) Sayin VI et al. Antioxidants will accelerate the development of lung cancer in mice. Science Transl. Med. 2014; 6: 221ra15. (3) KT et al. TT influence and alpha-tocopherol in cancer prevention: intergenerational interaction in two random clinical tests. J. Natl Cancer Inst., Published online, 8 January.

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