Scientists at North Carolina (US) University have found an enzyme, USP21, which affects the expansion of more aggressive breast cancer, called malnutrition or trivial, according to a survey found in Scotland. it was today published in the journal Cell.
This result could help development experts to develop new amendments to counteract unwanted offensive words of this kind. cancer breastfeeding
Indeed, one of the authors of the review, Michael Emanuele, believed that the enzyme “could represent a new and future focus for medicinal intervention”.
"We are also believe that we are going to hit the USP21 target to get cancer cells that could already be used routinely to treat patients with the condition. T this "said the expert.
In that type of cell, proteins called factors of control are controlled by the degree to which genetic information is copied from the DNA to a RNA (acid or ribonucleic acid) agent within the cells.
This transcript is fundamental to complete the cycle and the size of each cell, including cancer.
FOXM1 is one of the transcription factors, found in important forms of breast cancer type, although scientists cannot fully understand the strategies governing this process.
Writers of the research used a method of searching that is based on RNA's invasion to find out whether or not they are out about FOXM1 and found that the enzyme USP21 promoted the presence of the government and its stability.
The greater the number of that bone in the cells, the rest of FOXM1 was protected in the circle of cells.
As USP21 left, the scientists sought a larger reduction in the networked proteins involved in FOXM1 and delays in the progression of the circuit program.
The research shows that the decrease in the "mild" enzyme is cancerous cells of basal and bones to paclitaxel, a treatment for this type of disease, which scientists believe would play a crucial role. USP21 in diversification and healing. the ability of basal cancer, with high concentrations of the enzyme and the protein.
"We found that USP21 is usually regulated in basic, three-person cancer.", said Emanuele, who said it would be possible to develop a drug to prevent the future enzyme to "cancel" the cancer cells that removed FOXM1 and stop them from growing and developing.
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